Short-acting barbiturates

Similar questions:

Short-acting barbiturates are metabolized mainly in the

liver.
NDEB released questions set 3

The chief mechanism by which the body metabolizes short-acting barbiturates is

hydroxylation and oxidation.
NDEB released questions part 2 set 15

The chief mechanism by which the body metabolizes short-acting barbiturates is

hydroxylation and oxidation.
NDEB released questions set 3

The brief duration of general anesthetic action of an ultra-short-acting barbiturate is due to what factor?

Rapid rate of redistribution from the brain to peripheral tissues
Antianxiety agents and Antiepileptics

The length of hypnotic action of ultrashort-acting barbiturate after a single dose is

5 to 20 minutes
Antianxiety agents and Antiepileptics

Ultrashort-acting barbiturates

for induction of general anesthesia
Antianxiety agents and Antiepileptics

Short-acting barbiturates

for treating insomnia, can be given preoperatively, before a dental appointment, to allay anxiety
Antianxiety agents and Antiepileptics

Contraindications to the use of ultra-short-acting barbiturates for general anesthesia:
1. Porphyria
2. Liver dysfunction (they are metabolized in the liver)
3. Emphysema
4. Previous addiction to sedative hypnotic drugs

All of the above
Antianxiety agents and Antiepileptics

The chief mechanism by which the body metabolizes short-acting barbiturates is

hydroxylation and oxidation
Planets 33

Succinylcholine is a short-acting neuromuscular junction blocking agent useful for providing a brief paralysis to aid in intubating patients. It is short-acting because

it is subject to inactivation by plasma esterases
Autonomy

Speed of recovery from short-acting anesthesia with thiopental depends chiefly on the rapidity of

Redistribution from the brain to skeletal muscle
Sadative, Antidepressants

The action of the ultrashort-acting barbiturates is terminated primarily by the process of

Redistribution
Sadative, Antidepressants